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Abstract

BACKGROUND:Left -ventricular hypertrophy, Changes in vascular architecture, myo-cardial calcifications are main (structural abnormalities) of the heart that have been described in patients with (chronic kidney disease) and are the most common cause of(( mortality )).OBJECTIVE: To highlight ((Echo- cardiographic )) findings in children with different stages of chronic kidney disease (CKD) and (risk factors )  associated with abnormal echo findings.PATIENTS AND METHODS: A ((prospective study)), consist of 50 patients with chronic kidney disease, age ranged between (6 months - 14 years) admitted to ((nephrology- unit)) in children welfare teaching hospital in ((Medical- City complex)) during the period from (1st of Jul. 2019 to 30th of Jul. 2020) were included in the study. Detailed-history was taken, complete -physical examination, biochemical- investigations, Abdominal Ultra-sound, Chest- X Ray and Echo-cardiography- were done to all patients enrolled in the study.RESULTS:  The study included 29 (58%) male and 21 (42%) female, with (male: female ratio was1.4 : 1), The most common cause of CKD was renal abnormalities 25 (50%) most of them were ((reflux nephropathy and neurogenic bladder)).The most common abnormal Echo findings was (left -ventricular- hypertrophy) in 16 (32 %) of patients. There were no significant association between Echo findings and different stages ( III , IV , V ) of chronic kidney disease ( p > 0.05 ).                    There were significant association between (((abnormal Echo findings))) and some risk factors like- anemia and hypertension ( p ≤ 0.05 ).CONCLUSION: The most common cause of chronic kidney disease (CKD) was (((congenital renal abnormalities))) in which reflux -nephropathy and neurogenic- bladder is commonest. Echo-cardiogram reveal abnormal finding in 50% of children with CKD, most common abnormal echo finding was left ventricular hypertrophy (LVH). Anemia and hypertension were the most common risk factors that are associated with abnormal echo findings. 

DOI

10.52573/ipmj.2024.183406

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